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Facing
frightening situations is a part of life, and the brain is ready
to respond to such situations resulting the body 's
“fight-or-flight” response to ensure survival (Connie
& Rossouw, 2013).
Signs of this are expected as an adaptive reaction to intensely
stressful moments that eventually fade away after a short time,
without long term psychological consequences (Grawe,
2007).
Posttraumatic Stress Disorder (PTSD), however, differs in
intensity and duration, interfering in the person's normal daily
functioning (Fritzsche, McDaniel & Wirsching, 2014). PTSD is a
psychological injury, a reaction to the traumatic event and may be
developed when a person witnessed, personally experienced, or
learned that a relative or close friend has experienced a single
or repeated traumatic experience, with the threat of possible
injury or death (Romero, 2011). The emotions generated in such
cases are experienced as overwhelming for the human coping
mechanisms (Fritzsche, McDaniel & Wirsching, 2014).
Co-morbidity may also occur (Fritzsche, McDaniel & Wirsching,
2014) as trauma maximises the chances of other mental illnesses
developing (Fritzsche, Abbo,Frahm, Monsalve & Chen, 2014).
In
DSM-V, PTSD is found in Trauma and Stressor-Related Disorders
instead of in the previous Anxiety Disorders category of DSM-IV.
PTSD diagnostic criteria is established in the DSM-V as follows:
“Diagnostic
Criteria - 309.81
(F43.10)
Posttraumatic
Stress Disorder
Note:
The following criteria apply to adults, adolescents, and children
older than 6 years. For children 6 years and younger, see
corresponding criteria below.
A.
Exposure to actual or threatened death, serious injury, or sexual
violence in one (or more) of the following ways:
1.
Directly
experiencing the traumatic event(s).
2.
Witnessing, in
person, the event(s) as it occurred to others.
3.
Learning that
the traumatic event(s) occurred to a close family member or close
friend. In cases of actual or threatened death of a family member
or friend, the event(s) must have been violent or accidental.
4.
Experiencing
repeated or extreme exposure to aversive details of the traumatic
event(s) (e.g., first responders collecting human remains; police
officers repeatedly exposed to details of child abuse).
Note:
Criterion A4 does not apply to exposure through electronic media,
television, movie, or pictures, unless this exposure is work
related
B.
Presence of one
(or more) of the following intrusion symptoms associated with the
traumatic event(s), beginning after the traumatic event(s)
occurred:
1.
Recurrent,
involuntary, and intrusive distressing memories of the traumatic
event(s).
Note:
In children older than 6 years, repetitive play may occur in which
themes or aspects of the traumatic event(s) are expressed.
2.
Recurrent distressing dreams in which the content and/or affect
of the dream are related to the traumatic event(s).
Note:
In children, there may be frightening dreams without recognisable
content.
3.
Dissociative
reactions (e.g., flashbacks) in which the individual feels or acts
as if the traumatic event(s) were recurring. (Such reactions may
occur on a continuum, with the most extreme expression being a
complete loss of awareness of present surroundings.)
Note:
In children, trauma-specific re-enactment may occur in play.
4.
Intense or
prolonged psychological distress at exposure to internal or
external cues that symbolise or resemble an aspect of the
traumatic event(s).
5.
Marked
physiological reactions to internal or external cues that
symbolise or resemble an aspect of the traumatic event(s).
C.
Persistent
avoidance of stimuli associated with the traumatic event(s),
beginning after the traumatic event(s) occurred, as evidenced by
one or both of the following:
1.
Avoidance of or
efforts to avoid distressing memories, thoughts, or feelings about
or closely associated with the traumatic event(s).
2.
Avoidance of or
efforts to avoid external reminders (people, places,
conversations, activities, objects, situations) that arouse
distressing memories, thoughts, or feelings about or closely
associated with the traumatic event(s).
D.
Negative alterations in cognitions and mood associated with the
traumatic event(s), beginning or worsening after the traumatic
event(s) occurred, as evidenced by two (or more) of the following:
1.
Inability to
remember an important aspect of the traumatic event(s) (typically
due to dissociative amnesia and not to other factors such as head
injury, alcohol, or drugs).
2.
Persistent and
exaggerated negative beliefs or expectations about oneself,
others, or the world (e.g., “I am bad,” “No one can be
trusted,” “The world is completely dangerous,” “My whole
nervous system is permanently ruined”).
3.
Persistent,
distorted cognitions about the cause or consequences of the
traumatic event(s) that lead the individual to blame
himself/herself or others.
4.
Persistent
negative emotional state (e.g., fear, horror, anger, guilt, or
shame).
5.
Markedly diminished interest or participation in significant
activities.
6.
Feelings of
detachment or estrangement from others.
experience
happiness, satisfaction, or loving feelings).
E.
Marked
alterations in arousal and reactivity associated with the
traumatic
event(s),
beginning or worsening after the traumatic event(s) occurred, as
evidenced by two (or more) of the following:
1.
Irritable
behaviour and angry outbursts (with little or no provocation)
typically expressed as verbal or physical aggression toward people
or objects.
2.
Reckless or self-destructive behaviour.
3.
Hyper vigilance.
4.
Exaggerated
startle response.
5.
Problems with
concentration.
6.
Sleep
disturbance (e.g., difficulty falling or staying asleep or
restless sleep).
F.
Duration of the
disturbance (Criteria B, C, D, and E) is more than 1 month.
G.
The disturbance causes clinically significant distress or
impairment in social, occupational, or other important areas of
functioning.
H.
The disturbance is not attributable to the physiological effects
of a substance (e.g., medication, alcohol) or another medical
condition.
Specify
whether:
With
dissociative symptoms:
The individual’s symptoms meet the criteria for posttraumatic
stress disorder, and in addition, in response to the stressor, the
individual experiences persistent or recurrent symptoms of either
of the following:
1.
Depersonalisation:
Persistent or recurrent experiences of feeling detached from, and
as if one were an outside observer of, one’s mental processes or
body (e.g., feeling as though one were in a dream; feeling a sense
of unreality of self or body or of time moving slowly).
2.
Derealization:
Persistent or recurrent experiences of unreality of surroundings
(e.g., the world
around the individual is experienced as unreal, dreamlike,
distant, or
distorted).
Note:
To use this subtype, the dissociative symptoms must not be
attributable to
the
physiological effects of a substance (e.g., blackouts, behaviour
during alcohol
intoxication)
or another medical condition (e.g., complex partial seizures).
Specify
if:
With
delayed expression:
If the full diagnostic criteria are not met until at least 6
months after the event (although the onset and expression of some
symptoms may be immediate).
Posttraumatic
Stress Disorder for Children 6 Years and Younger
A.
In children 6 years and younger, exposure to actual or threatened
death, serious injury, or sexual violence in one (or more) of the
following ways:
1.
Directly
experiencing the traumatic event(s).
2.
Witnessing, in
person, the event(s) as it occurred to others, especially primary
caregivers.
Note:
Witnessing does not include events that are witnessed only in
electronic media, television, movie, or pictures.
3.
Learning that
the traumatic event(s) occurred to a parent or care giving figure.
B.
Presence of one (or more) of the following intrusion symptoms
associated with the traumatic event(s), beginning after the
traumatic event(s) occurred:
1.
Recurrent,
involuntary, and intrusive distressing memories of the traumatic
event(s).
Note:
Spontaneous and intrusive memories may not necessarily appear
distressing and may be expressed as play re-enactment.
2.
Recurrent
distressing dreams in which the content and/or affect of the dream
are related to the traumatic event(s).
Note:
It may not be possible to ascertain that the frightening content
is related to the traumatic event.
3.
Dissociative reactions (e.g., flashbacks) in which the child
feels or acts as if the traumatic event(s) were recurring. (Such
reactions may occur on a continuum, with the most extreme
expression being a complete loss of awareness of present
surroundings.) Such trauma-specific re-enactment may
occur
in play.
4.
Intense or
prolonged psychological distress at exposure to internal or
external cues that symbolise or resemble an aspect of the
traumatic event(s).
5.
Marked physiological reactions to reminders of the traumatic
event(s).
C.
One (or more) of
the following symptoms, representing either persistent avoidance
of stimuli associated with the traumatic event(s) or negative
alterations in cognitions and mood associated with the traumatic
event(s), must be present, beginning after the event(s) or
worsening after the event(s):
Persistent
Avoidance of Stimuli
1.
Avoidance of or
efforts to avoid activities, places, or physical reminders that
arouse recollections of the traumatic event(s).
2.
Avoidance of or efforts to avoid people, conversations, or
interpersonal situations that arouse recollections of the
traumatic event(s).
Negative
Alterations in Cognitions
1.
Substantially
increased frequency of negative emotional states (e.g., fear,
guilt, sadness, shame, confusion).
2.
Markedly
diminished interest or participation in significant activities,
including constriction of play.
3.
Socially
withdrawn behaviour.
4.
Persistent
reduction in expression of positive emotions.
D.
Alterations in
arousal and reactivity associated with the traumatic event(s),
beginning or worsening after the traumatic event(s) occurred, as
evidenced by two (or more) of the following:
1.
Irritable
behaviour and angry outbursts (with little or no provocation)
typically expressed as verbal or physical aggression toward people
or objects (including extreme temper tantrums).
2.
Hypervigilance.
3.
Exaggerated
startle response.
4.
Problems with
concentration.
5.
Sleep
disturbance (e.g., difficulty falling or staying asleep or
restless sleep).
E.
The duration of
the disturbance is more than 1 month.
F.
The disturbance
causes clinically significant distress or impairment in
relationships with parents, siblings, peers, or other caregivers
or with school behaviour.
G.
The
disturbance is not attributable to the physiological effects of a
substance (e.g., medication or alcohol) or another medical
condition
Specify
whether:
With
dissociative symptoms:
The individual’s symptoms meet the criteria for posttraumatic
stress disorder, and the individual experiences persistent or
recurrent symptoms of either of the following:
1.
Depersonalisation:
Persistent or recurrent experiences of feeling detached from, and
as if one were an outside observer of, one’s mental processes or
body (e.g., feeling as though one were in a dream; feeling a sense
of unreality of self or body or of time moving slowly).
2.
Derealization:
Persistent or recurrent experiences of unreality of surroundings
(e.g., the world around the individual is experienced as unreal,
dreamlike, distant, or distorted).
Note:
To use this subtype, the dissociative symptoms must not be
attributable to the physiological effects of a substance (e.g.,
blackouts) or another medical condition (e.g., complex partial
seizures).
Specify
if:
With
delayed expression:
If the full diagnostic criteria are not met until at least 6
months after the event (although the onset and expression of some
symptoms may be immediate).” (First, 2014,
309.81-
F43.10).
Concerning
the prevalence of PTSD, DSM-V states that it may vary depending of
culture, developmental stage, demography and type of risks
involved in the person's profession (First, 2014). In Australia, 5
to 10% of the adults, and 1 to 6 % of children are expected to
suffer PTSD in their life-time (Australian
Centre for Posttraumatic Mental Health, 2013). Additionally,
“6% of women may suffer PTSD after obstetric procedures”
(Cockburn & Pawson, 2007). Comorbidity is found in 86% of
males and 77% of females (Australian Centre for Posttraumatic
Mental Health, 2013). Research affirmed that in the United States,
the prevalence of PTSD throughout lifetime is 8%, while in
Germany, it is 1.3% and in Algeria 37.4% (Davidson,
2001).
Survivors of war and rape are among the highest percentage, while
elderly adults present the lowest percentage (First, 2014). A
study utilising a sample of 379 respondents who survived the
attack on the World Trade Centre on September 11 revealed the
prevalence of PTSD in more than a third of those who were directly
exposed, 20% in those who just witnessed the traumatic events on
that day, and 35% of those who learned that a close relative or
friend was directly exposed to the events (Worcester, 2011).
Moreover,
Cuffe, Cuffe, Addy, Garrison and Waller's (1998) research
concerning the prevalence of PTSD in 490 older adolescents ranging
from 16 to 22 year of age and from different ethnic groups
demonstrated that a fraction of 49.37% female suffered rape or
sexual abuse in childhood, and 85.02% were witness to a traumatic
incident (Cuffe, Cuffe, Addy, Garrison & Waller, 1998). PTSD
was found frequently in those who suffered rape or were victims of
crime (Cuffe, Cuffe, Addy, Garrison & Waller, 1998). Family
attachment and living with the family or by themselves were not
considered to raise the risk of PTSD development (Cuffe, Cuffe,
Addy, Garrison & Waller, 1998). In addition, Ditlevsen and
Elklit's (2012) study showed that gender and type of trauma
differences influence PTSD prevalence. From 5520 subjects
interviewed, 20.6% or 1075 participants, 25.6% of which were
female and 13.2% males, met the DSM criteria for PTSD (Ditlevsen &
Elklit, 2012). Due to gender inherent differences, females
demonstrated twice the number of incidences of PTSD than males
(Ditlevsen & Elklit, 2012). “Disaster and accident” were
the type with the “highest gender difference”, while “violence
was the lowest gender difference”, but the highest PTSD
prevalence (Ditlevsen & Elklit, 2012). Females are more
susceptible, firstly, to “disaster and accident” types of
trauma, and secondly, to “chronic illness and loss”(Ditlevsen
& Elklit, 2012).
Clinicians
find PTSD presentation of symptoms in “acute, chronic, delayed,
complex, or even masked forms” (McHugh
& Treisman, 2007). The type and duration of symptoms indicate
the severity level (McHugh & Treisman, 2007). Soon after an
unexpected traumatic incident, the person may act as if what
happened was not real, feel agitated, and not eating or sleeping
well during the first hours or days after the event (McHugh &
Treisman, 2007). Following this, the person will recall the event
with high anxiety, may feel “sadness, fear, guilt, anger,
irritation, numbness, depersonalisation, hypervigilance, have
nightmares and flashbacks” and avoid what is
connected
with the trauma (Australian Centre for Posttraumatic Mental
Health, 2013). Dave & Mehta's (2008) research indicated that
women with PTSD show more low energy, anxiety, depression and
suicidal ideation than men.
During
the interview, some clients find it difficult to begin speaking
about the traumatic event aiming to avoid the “horror and
helpless” feelings, which arise shortly after they begin to
report the incident (Yehuda, 2002). Others show disengagement from
the conversation or clearly refuse to talk about the subject,
which does not mean that the client is unwilling to cooperate with
the treatment, but instead, it is a sign that talking about the
trauma causes extreme distress (Yehuda, 2002). Some become very
agitated or show pessimism and a “negative mood”. Moreover,
being unable to arrive on time for the session due to lack of
organisation, forgetting the address, “emotional absence or
detachment”, as well as forgetting parts of the episode, are
examples of dissociation (Yehuda, 2002). In general, the client's
family may often complain that their relationship becomes
“superficial and distant” (Yehuda, 2002). As PTSD impacts the
brain's function not only during the event, but also during the
reexperiencing as a result of the activation of the “limbic-system
and pre-frontal shuts down”, the client experiences difficulty
in verbally expressing themselves, processing new information,
comprehending and “finding meaning” for what occurred
(Hartley, 2009).
The
mental heath professional must note that PTSD symptoms such as
‘‘difficulty falling or staying
asleep, difficulty concentrating, diminished interest or
participation in significant activities,
restricted range of affect and irritability can also be caused by
other disorders instead of the related trauma” (McHugh, &
Treisman, 2007). Clinicians
may also find cases of malingering (when the person intentionally
fakes their PTSD symptoms in order to receive financial
compensation) and factitious (when the person misunderstands their
conceptualisation of their traumatic event) presentations of PTSD
symptoms (Rosen
& Taylor, 2007).
Although
it is known that the most common symptoms of PTSD are
“reexperiencing, avoidance/numbing, and hyperarousal”
(Cloitre, Courtois, Charuvastra, Carapezza, Stolbach, & Green,
2011), DSM-V states that the predominance of “clinical
presentation varies”(First, 2014). For instance, while some
people present more “emotional and behavioural symptoms with
fear-based re-experiencing, others show anhedonic or dysphoric
mood states and negative cognitions”(First, 2014). Some display
more “arousal and reactive-externalising symptoms, while others
show, dissociative symptoms”(First, 2014). The combination of
these symptoms can also be found (First, 2014). Further studies
are needed to know how to differentiate and better understand the
effects caused by pre and post-migration stressors, acculturation,
chronic grief, discrimination and other difficulties experienced
by immigrants and refugees upon PTSD symptom presentation.
For
instance, Davidson's
(2001) research
established a high percentage of somatization and irritable bowel
syndrome in people diagnosed with PTSD.
Furthermore, the intense period of fight or flight survival
reaction when the person is experiencing the trauma, causes
muscles and joints to become very tense, the body memorises and
keeps repeating this reaction, causing long term muscles and joint
pain, which must be separated from the “physiological effects of
a substance (e.g., medication, alcohol) or another medical
condition” stated in criteria H (First, 2014). Galovski, Mott,
Young-Xu and Resick's (2011) study concerning “gender
differences in the clinical presentation of PTSD” with 162 woman
and 45 man who suffered sexual assault or physical violence
indicated that a similar percentage of females and males feel
guilt, however, males feel more anger while females show more
“health related symptoms”. The current results of Carleton,
Mulvogue and Duranceau's (2014) study with 129 women who
experienced spouse violence stated that personality attributes
cause more “comorbid symptoms”, thus, influencing PTSD
symptom-presentation.
Adolescents
present symptoms of reexperiencing such as “intrusive thoughts
and nightmares, amnesia, detachment, sleep disturbance and
hypervigilance, refusing to talk about the trauma and withdrawal
from friends and activities” (Perkin, 2008). Those with chronic
symptoms demonstrate “dissociative symptoms such as
derealization, depersonalisation, self-harm, substance abuse, and
intermittent angry or aggressive outbusters” (Perkin, 2008).
Primary school children principally exhibit “re-enactment of the
trauma through play, speech and drawing, instead of amnesia,
avoidance, numbness and flashbacks” (Perkin, 2008). Toddlers
manifest “generalised anxiety as separation fears, strangers
anxiety, fear of monsters or animals, as well as sleep
disturbance, and avoidance of situations, words or symbols that
may be related to the trauma” (Perkin, 2008).
For
example, in Green, and colleagues' (1991) study with 600 adults
and children aged 2 to 15years of age, two years after a flood
experience, PTSD intrusion symptoms” such as “distressing
dreams”, had the highest percentage of occurrence in children,
followed by “low affect, irritability and anger outburst”
(Green, Korol, Grace, Vary, Leonard, Glesser, &
Smitson-Cohen, 1991). PTSD symptoms were found in all age groups
with fewer occurrences in the youngest children (Green, Korol,
Grace, Vary, Leonard, Glesser, & Smitson-Cohen, 1991). Gender,
threat of death, parent’s depression and irritability were also
contributing factors to children's PTSD development (Green,
Korol, Grace, Vary, Leonard, Glesser, & Smitson-Cohen, 1991).
Another
common symptom found in people with PTSD are “flashbacks”
(First, 2014). These are “dissociative symptoms” where the
person feels through sensory or visual organs, intrusive pieces of
the trauma experienced as if it were happening again, even though
they are still aware of their current reality (First, 2014). It
may last from seconds to days, causing long term “distress and
arousal” (First, 2014). Children under six re-live the trauma
through playing or “dissociative states” (First, 2014).
Criteria C1 of DSM-V, “avoidance, is achieved when the person
distract themselves in order to avoid thoughts, memories,
feelings, or talking about the traumatic event”, and C2, when
avoiding “activities, objects, situations, or people who remind
them of the event” (First, 2014).
One
of the “negative alterations” symptoms listed in criteria D1
is called “amnesia” (First, 2014). This “inability
to remember” specific moments of their past experiences was
found in Graham, Herlihy and Brewin's (2014) study in which 38
asylum seekers and refugees diagnosed with PTSD, from 19 different
countries were interviewed. The study also highlighted that
refugees' visas have been denied by several countries' agencies as
refugees with PTSD are unable to recall past life or trauma
moments, which is a requirement in order to prove credibility of
their situation and need for a refugee visa (Graham, Herlihy &
Brewin, 2014).
Another
significant factor that is generally overlooked by clinicians is
the comorbidity of PTSD and nicotine (Stewart, 2014). Stewart's
(2014) study with 604 people who were 41 years old and diagnosed
with PTSD found that smoking is used by them as self-medication in
order to decrease stress levels (Stewart, 2014). Additionally,
smokers diagnosed with PTSD are less willing to quit smoking as
their energy seems to be needed to deal with PTSD symptoms rather
than facing smoking withdrawal symptoms, which compromises their
overall health and affects positive outcomes in reducing PTSD
symptoms (Stewart, 2014).
Regarding
impacts, PTSD has been known as a common disorder in many
countries, negatively affecting peoples lives for an average of 20
years (McHugh & Treisman, 2007). In the United States,
research has revealed that, as people with PTSD do not seek mental
health treatment, only 4% receive the appropriate diagnosis, with
19% attempting suicide (Davidson, 2001). Moreover, there is a loss
of 3 billion dollars a year, with one work day a week affected by
the absence of workers with PTSD symptoms (Davidson, 2001), which,
therefore, increases unemployment
by 150%
and causes
instability in 60% of marriage relationships (Galovski
& Lyons, 2004).
PTSD
is also considered a cause of increasing the “risk
for depression, anxiety, alcohol or substance use disorders,
hypertension, bronchial asthma, peptic ulcer, diseases of the
cardiovascular, digestive, musculoskeletal, endocrine,
respiratory, and nervous systems, as well as infectious diseases”
(Davidson,
2001). Those
who were abused in childhood have a high percentage of having PTSD
(Davidson, 2001) and consequently, a higher percentage of
“smoking,
contracting sexually transmitted diseases, developing severe
obesity, ischemic heart disease, cancer, chronic obstructive
pulmonary disease, fractures, and liver disease than the general
population”(Davidson,
2001).
Galovski
and Lyons's (2004) review of 141
articles indicate that
“secondary traumatization” is one of the impacts caused by
“numbing/arousal symptoms” of PTSD on the relationship of
veterans with their families. The trauma can also be passed to the
next generation, when a child becomes traumatised by learning of
their parent's trauma, or copies their symptoms to identify with
them (Galovski & Lyons, 2004). Similarly, Ray and Vanstone's
(2009) study with 10
“contemporary
peacekeepers”
from 37 to 46 years old, made
evident that “veteran's anger” and “emotional withdrawal
from family support” not only impacts their family
relationships, but prevents positive treatment outcomes, and,
hence, are the first symptoms that should be addressed during PTSD
treatment (Ray & Vanstone, 2009).
A
thorough assessment of PTSD includes the collection of the
relevant data in order to assess presence and severity of the
symptoms as well as diagnostic criteria, trauma exposure and
comorbidity (Frueh, Grubaugh, Elhai & Ford, 2012). The
clinician must also explore the client's history, “marital
status and family situation, the client's strengths and
resilience, and response to previous treatments” (Frueh,
Grubaugh, Elhai & Ford, 2012). Furthermore, the clinician also
need to make a differential diagnosis and prognosis, decide the
best approach to be used, establish treatment goals and plan, and
monitor the treatment and its outcomes (Frueh, Grubaugh, Elhai &
Ford, 2012). This interview can take from 15 to 60 minutes, where
the Mental Status Examination is applied, and suicide assessment
may be necessary (Frueh, Grubaugh, Elhai, & Ford, 2012).
During the PTSD assessment process, the mental health professional
need carefully eliminate the possibility of “malingering” or
other forms of “factitious-PTSD”, (Rosen & Taylor, 2007).
The
National Centre for Posttraumatic Stress Disorder, suggests the
use of the multimethod approach for the assessment (Mueser,
Rosenberg, & Rosenberg, 2009). According
to the “multimethod
approach”, the
most common methods and measures that can be used to screen,
diagnose and monitor treatment outcomes are “Diagnostic
Interviewing, Self-report questionnaires and Psychophysiology
measures”(Friedman, Keane & Resick, 2014). Furthermore, the
clinician may need to use “multiscale personality inventories”,
see the client's medical records and collect information related
to client's behaviour from other people (Friedman, Keane &
Resick, 2014).
Different
measures can be used for different populations, considering
different “cultural and linguistic background and different
cultural trauma conceptualisations” (Australian Centre for
Posttraumatic Mental Health, 2013).
An
adequate treatment of PTSD involves building a strong therapeutic
alliance (more time will be required for clients who have suffered
more prolonged exposure or severe traumas), ensuring the client's
safety, making a careful “assessment, case formulation,
considering PTSD chronicity, comorbidity, and cognitive and
physical health, including hormone levels which are affected
during the stress reaction” (Martin & Captain, 2013). In
addition, the therapist should consider the client's background,
treatment expectancy, treatment setting (including environments
where the client is exposed to continuous stress and trauma such
as detention centres, refugee camps, corrective
centres, theatres of combat, women's refuges), individualised
treatment planning, and monitoring (Australian Centre for
Posttraumatic Mental Health, 2013). The clinician may also need to
provide practical information, psycho-education and foster
client's network support and encourage regular physical exercises
(Australian Centre for Posttraumatic Mental Health, 2013). Most
importantly, clinicians must be skilled, trained, and able to use
their clinical judgement to chose the best approach to use as a
“first or second line intervention” (Forbes, Creamer, Bisson &
Cohen, 2010).
Treatments
may combine psychological and medical interventions, where the
most indicated are the trauma-focus interventions, in order to
reduce PTSD symptoms, anxiety and depression (Australian Centre
for Posttraumatic Mental Health, 2013). Medication should not be
used as a frontline treatment, but only after one month of
symptoms appearance, and when psychological treatment is
unsuccessful, or when it is necessary to treat comorbidity
symptoms (Australian Centre for Posttraumatic Mental Health,
2013). If medication for adults with PTSD is needed, “selective
serotonin re-uptake inhibitor (SSRI) antidepressants” are the
most prescribed (Australian Centre for Posttraumatic Mental
Health, 2013). It is important to highlight that debriefing is not
advised soon after the trauma to prevent client's
re-traumatisation (Australian Centre for Posttraumatic Mental
Health, 2013).
“Trauma-focused
Cognitive-Behavioural Therapy (TFCBT), Eye Movement
Desensitisation and Reprocessing (EMDR), Psychodynamic
Psychotherapy, Supportive Counselling/Therapy, Narrative Exposure
Therapy (NET), Hypnotherapy, and Psychological Debriefing are the
common approaches used to treat PTSD” (Australian Centre for
Posttraumatic Mental Health, 2013). However, Bisson,
Roberts, Andrew, Cooper and Lewis's (2013) review of 70
studies including 4761 people about
Psychological Therapies for PTSD, concluded that “Individual
Trauma-Focused Cognitive Behavioural Therapy (TF-CBT)
and Eye
Movement Desensitisation and Reprocessing (EMDR)”
are the more empirical validated, dominant, and effective
treatments recommended for PTSD in adults.
TF-CBT
uses techniques from Cognitive Therapy and Behavioural Therapy
intending to change people's thoughts and behaviours in order to
better deal with the traumatic
incidents by the exposure of those
memories (Bisson,
Roberts, Andrew, Cooper & Lewis, 2013).
EMDR is the progressive recall of trauma memories while making eye
movements when the person reprocesses and replaces the trauma
memories for more adaptive ones, decreasing stress levels (Bisson,
Roberts, Andrew, Cooper, & Lewis, 2013).
For children and adolescents,“developmentally
appropriate Trauma-Focussed Cognitive Behavioural Therapy” is
the most recommended treatment, including parent's involvement,
children's homework, the use of “validated protocols”,
attractive colourful materials and media (Australian Centre for
Posttraumatic Mental Health, 2013). Further
investigation about effectiveness of psychodynamic psychotherapy,
supportive counselling/therapy, narrative exposure therapy (NET),
hypnotherapy, and psychological debriefs are needed (Australian
Centre for Posttraumatic Mental Health, 2013).
As
attachment and control are basic human needs, establishing a
trustful therapeutic relationship and ensuring constant safety are
the first steps to build a strong therapeutic alliance and
facilitate change (Connie
& Rossouw, 2013).
Clients with PTSD can be more resistant to treatment as their
reactions are in the “survive mode”(Conte, 2009). Those with
insecure or disorganised attachment may carry a degree of
developmental trauma, which enhances PTSD symptoms (Hartley,
2009). Hence, the human encounter must be genuine, individualised
and non-judgemental so that the client can have a restorative
emotional experience (Hartley, 2009). Clinicians also need to
demonstrate empathy, acceptance, unconditional positive regard,
recognise their counter transferences, and ability to withstand
the feelings contained in the client's lived experience concerning
the catastrophic event they suffered (Hartley, 2009). In addition,
an ability to apply effective treatment methods, basic and
advanced skills such as active listening, attending behaviour,
questioning, immediacy, reflecting, paraphrasing, and summarising
are required (Sommers-Flanagan & Sommers-Flanagan, 2012).
Moreover, “Self-awareness, multicultural knowledge, and
culturally specific techniques, which are multicultural
competences” should also be considered (Sommers-Flanagan &
Sommers-Flanagan, 2012). Furthermore, the clinician's supervision,
and self-care are crucial to prevent secondary traumatisation
(Stender, 2013).
In
conclusion, PTSD is a common and very distressing clinical
condition that occurs after a traumatic event, impacting people's
lives, and their families, in many areas (McHugh
& Treisman, 2007).
Type,
presentation and duration of symptoms vary and must be
differentiated from other disorders (McHugh & Treisman, 2007).
Assessment measures and treatment approaches should be culturally
appropriate (Australian Centre for Posttraumatic Mental Health,
2013). A trustful and genuine therapeutic relationship, in a safe
environment, are the key elements to assist clients with PTSD to
rebuild their lives (Blackwell, 2005). Further studies for a
better comprehension of the correlation of PTSD symptoms and
refugee's and immigrants' holistic experience is needed.
Mrs Glaucia Barbosa, PACFA Reg. Provisional 25212 MCouns, MQCA(Clinical) ABN: 19 476 932 954
References
Australian
Centre for Posttraumatic Mental Health. (2013).
Australian
guidelines for the treatment of acute stress disorder &
posttraumatic stress disorder. ISBN:
978-09923138-7-6.Online: 978-09923138-8-3. Retrieved from:
http://www.acpmh.unimelb.edu.au/
Bisson,
J.I., Roberts, N.P., Andrew, M., Cooper, R., & Lewis, C. (2013).
Psychological therapies for chronic post-traumatic stress disorder
(PTSD) in adults. Cochrane
Depression, Anxiety and Neurosis Group. The
Cochrane
Collaboration: Published by John Wiley & Sons,Ltd. doi: 10.1002/14651858.CD003388.pub4.
Black,
D.W., & Grant, J.E. (2014).DSM-5
guidebook: The essential companion to the diagnostic and statistical
manual of mental disorders. 5th
edition.
Arlington, VA: America Psychiatry Publishing.
Blackwell,
D. (2005). Counselling
and psychotherapy with refugees.
London: J.Kingsley Publishers.
Carleton,
R. N., Mulvogue, M. K., & Duranceau, S. (2014). PTSD personality
subtypes in women exposed to intimate-partnerviolence. Psychological
Trauma: Theory, Research, Practice, and Policy,
No Pagination Specified.doi:10.1037/tra0000003
Cloitre,
M., Courtois, C. A., Charuvastra, A., Carapezza, R., Stolbach, B. C.,
&Green, B. L. (2011). Treatment of complex PTSD: Results of the
ISTSS expert clinician survey on best practices.Journal
of Traumatic Stress, 24(6),
615-627. doi:10.1002/jts.20697
Cockburn,
J., & Pawson, M. E. (2007). Psychological
challenges in obstetrics and gynaecology: The clinical management
. London : Springer
Connie,
H., & Rossouw, P.(2013). BrainWise
Leadership.
Sydney,AU: Learning Quest.
Conte,
C. (2009). Advanced
Techniques for Counselling and Psychotherapy.
USA: Springer Publishing Company,
LLCCuffe,
S. P., Cuffe, S. P., Addy, C. L., Garrison, C. Z., & Waller, J.
L. (1998). Prevalence of PTSD in a community sample of older
adolescents. Journal of
the American Academy of Child and Adolescent Psychiatry, 37(2),
147; 147-154; 154.
Dave,
P. & Mehta, M. (2008).Mental Health and Women. Important
correlation. India: Kelpaz Publications.
Davidson,
J.R.T., (2001). Recognition
and treatment of posttraumatic stress disorder JAMA.286(5):584-588.
doi:10.1001/jama.286.5.584.
Ditlevsen,
D.N., & Elklit, A. (2012). Gender, trauma type, and PTSD
prevalence: A re-analysis of 18 Nordic convenience samples. Annals
of General Psychiatry.
11:26 doi:10.1186/1744-859X-11-26
First,
M. B. (2014). DSM-5
handbook of differential diagnosis. Trauma- and Stressor-Related
Disorders, (309.81-F43.10).
Arlington,
Va: American Psychiatric Publishing. doi:
10.1176/appi.books.9780890425596.991543
Forbes,
D., Creamer, M., Bisson,J. I., & Cohen, J. A. (2010). A guide to
guidelines for the treatment of PTSD and related conditions. Journal
of Traumatic Stress, 23(5),
537; 537-552; 552.doi: 10.1002/jts.20565
Friedman,
M. J., Keane, T. M., & Resick, P. A.(2014). Handbook
of PTSD. Science and Practice. USA:
The Guilford Press.
Friedman,
M. J., Resick, P. A., Bryant, R. A., & Brewin, C. R. (2011).
Considering PTSD for DSM-5.Depression and Anxiety, 28(9),
750-769. doi:10.1002/da.20767
Fritzsche,
K., Abbo,
C., Frahm, G., Monsalve, S.D., & Chen, F.K
(2014).
Psychosomatic
Medicine. An International Primer for the Primary Care Setting. New
York,
USA:Springer.
Fritzsche,
K., McDaniel, S.H., & Wirsching, M. (2014). Psychosomatic
Medicine. An International Primer for the Primary Care Setting. New
York, USA:Springer.
Frueh,
B.C., Elhai, J.D., Grubaugh,A.L., Monnier, J., Kashdan, T.B.,
Sauvageot, J.A. et
al.(2005).
Documented combat exposure of US veterans seeking treatment
for combat-related post-traumatic stress disorder.
British
Journal of Psychiatry, 186, pp. 467–472
Frueh,
C., Grubaugh, A., Elhai, J. D., &Ford, J. D. (2012). Assessment
and treatment planning for PTSD
(1st
ed.). Hoboken: Wiley.
Fulford,
K., Davies, M., Gipps,R., Graham, G., Sadler,J., Stanghellini,G., &
Thornto, T. (2013). The
Oxford handbook of philosophy and psychiatry.UK:
Oxford University Press.
Galovski,
T., & Lyons, J. A. (2004). Psychological sequelae of combat
violence: A review of the impact of PTSD on the veteran's family and
possible interventions. Aggression
and Violent Behaviour, 9(5),
477-501. doi:http://dx.doi.org/10.1016/S1359-1789(03)00045-4
Galovski,T.
E., Mott, J.,Young-Xu, Y., & Resick, P. A. (2011). Gender
differences in the clinical presentation of PTSD and its concomitants
in survivors of interpersonal assault. J
Interpers
Violence 2011
26: 789. doi: 10.1177/0886260510365865
Graham,
B., Herlihy, J., & Brewin, C. R. (2014). Over general memory in
asylum seekers and refugees. Journal
of Behaviour Therapy and Experimental Psychiatry, 45(3),
375-380.
doi:http://dx.doi.org.ezproxy.library.uq.edu.au/10.1016/j.jbtep.2014.03.001
Grawe,
K. (2007). Neuropsychotherapy.
How The Neuroscience Inform Effective Psychotherapy.
New
York-USA: Taylor and Francis Group.
Green, B. L., Korol, M., Grace, M. C., Vary, M. G., Leonard, A. C., Gleser, G. C., & Smitson-Cohen, S. (1991). Children and disaster: Age, gender, and parental effects on PTSD symptoms. Journal of the American Academy of Child & Adolescent Psychiatry, 30(6), 945-951.doi:http://dx.doi.org/10.1097/00004583-199111000-00012Hartley, L. (2009). Contemporary body psychology. The Chiron Approach. USA: Routledge.Kaplan, A. (2009). DSM-V controversies. Psychiatric Times, 26(1), 1-10.Martin, P. R., Captain. (2013). Hormone treatment for PTSD. United States Naval Institute. Proceedings, 139(8), 79-80.
McHugh,
P. R., & Treisman, G. (2007). PTSD: A problematic diagnostic
category. Journal
of Anxiety Disorders, 21(2),
211-222.
Mueser,
K. T., Rosenberg, S. D., & Rosenberg, H. J. (2009). Assessment
of trauma and posttraumatic disorders. (pp. 37-52). Washington,
DC, US: American
Psychological Association. doi:10.1037/11889-002
Perkin,
R. M. (2008). Paediatric hospital
medicine: Textbook of inpatient management.
Philadelphia, PA: Wolters Kluwer.
Ray,
S. L., & Vanstone, M. (2009). The impact of PTSD on veterans’
family relationships: An interpretative phenomenological inquiry.
International
Journal of
Nursing Studies, 46(6),
838-847. doi:http://dx.doi.org/10.1016/j.ijnurstu.2009.01.002
Resnick,P.J.
(2003). Guidelines for evaluation of malingering in PTSD.
Posttraumatic stress disorder in litigation: guidelines for forensic
assessment. pp.187–205.
Washington, DC: American Psychiatric Press.
Romero,
D. (2013). Ptsd. Museums
& Social Issues, 6(2),
161; 161-167; 167.
doi: ttp://dx.doi.org.ezproxy.library.uq.edu.au/10.1179/msi.2011.6.2.161
Rosen,
G. M., & Taylor, S. (2007). Pseudo-PTSD. Journal
of Anxiety Disorders, 21(2),
201-210. doi:http://dx.doi.org.ezproxy.library.uq.edu.au/10.1016/j.janxdis.2006.09.011
Sommers-Flanagan,
J., & Sommers-Flanagan, R. (2012). Counselling and
psychotherapy theories in context and practice: Skills, strategies,
and techniques
(2nd ed.). Hoboken: Wiley.
Stender,
J. L. (2013). Counsellor
perceptions of self-care: Techniques and strategies.
(Ph. D., State University of New York at Buffalo). ProQuest Dissertations
and Theses,. (1511026744).
Stewart,
T. C. (2014). Nicotine
and PTSD symptom duration.
(Ph.D., Yeshiva University). ProQuest
Dissertations and Theses,.
(1547746049).
Yehuda,
R. (2002). Treating trauma survivors
with PTSD. American Psychiatric
Publishing, Inc. ISBN: 1585620106, 9781585620104, 9781585627844 Pages:
216
Worcester,
S. (2011). Study challenges PTSD prevalence post sept. 11. Clinical
Psychiatry News, 39(10), 18; 18-18; 18.
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